Miguel E. Habeych, MD, MPHAssistant Professor of Neurological Surgery/Molecular Genetics and Biochemistry
Director, Center for Clinical Neurophysiology
Miguel E. Habeych, MD, MPH joined the Department of Neurological Surgery in September of 2005 as associate director of the Center for Clinical Neurophysiology. He was appointed director of the center in March of 2008.
Dr. Habeych attended medical school and did his internship at the Universidad Industrial de Santander (Colombia), obtaining his medical degree in 1989. He completed training in clinical neurology at the National University of Colombia, graduating in 1995.
After practicing general neurology and electroencephalography for three years, he pursued a master of public health degree at the Graduate School of Public Health of the University of Pittsburgh, graduating in 2000. He did a postdoctoral fellow research with the Center for Education and Drug Abuse Research (CEDAR) of the University of Pittsburgh’s School of Pharmacy, and worked there as a research associate from 2001 to 2004.
Dr. Habeych completed a fellowship in clinical neurophysiology/intraoperatory neurophysiological monitoring at the Center for Clinical Neurophysiology in July of 2005.
Dr. Habeych's publications can be reviewed through the National Library of Medicine's publication database.
Children’s Hospital of Pittsburgh of UPMC
Magee-Womens Hospital of UPMC
UPMC St. Margaret
Veterans Affairs Pittsburgh Health Care System, Oakland
Western Psychiatric Institute and Clinic
Professional Organization Membership
Colombian College of Neuropharmacology
Colombian Association of Neurology
The American Medical Association
American Society of Neurophysiological Monitoring
American Clinical Neurophysiology Society
Charles F. Reynolds Medical History Society
Miguel E. Habeych, MD, MPH, just finished a research effort on clarifying the role of Botulinum neurotoxin (BtNtx) pre-treatment in the intraoperative neurophysiological monitoring (IONM) of patients who were going to have a microvascular decompression procedure, because, suffered a Hemifacial Spasm (HFS). The results showed that the abnormal muscular response obtained in patients with HFS after stimulating the facial nerve, the so-called “lateral spread response” (LSR), is different in amplitude in these two group of patients with or without previous BtNtx treatment. In fact, Dr. Habeych found that the LSR is more robust in patients with previous treatments with BtNtx, and therefore, it confounds their IONM, because, they give the false sensation of presence. The reason for this response to be higher in amplitude seems to be the disorganized re-innervation induced by the BtNtx.